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A de novo transcriptome analysis showsthat modulation of the JAK-STAT signalingpathway by salmonid alphavirus subtype 3favors virus replication in macrophage/dendritic-like TO-cells

dc.contributor.authorXu, Cheng
dc.contributor.authorEvensen, Øystein
dc.contributor.authorMunangandu, Hetron Mweemba
dc.date.accessioned2016-07-19T07:45:59Z
dc.date.accessioned2016-09-06T14:01:56Z
dc.date.available2016-07-19T07:45:59Z
dc.date.available2016-09-06T14:01:56Z
dc.date.issued2016
dc.identifier.citationBMC Genomics 2016, 17nb_NO
dc.identifier.issn1471-2164
dc.identifier.urihttp://hdl.handle.net/11250/2404766
dc.description-nb_NO
dc.language.isoengnb_NO
dc.rightsNavngivelse 3.0 Norge*
dc.rightsNavngivelse 3.0 Norge*
dc.rights.urihttp://creativecommons.org/licenses/by/3.0/no/*
dc.titleA de novo transcriptome analysis showsthat modulation of the JAK-STAT signalingpathway by salmonid alphavirus subtype 3favors virus replication in macrophage/dendritic-like TO-cellsnb_NO
dc.typeJournal articlenb_NO
dc.typePeer reviewednb_NO
dc.date.updated2016-07-19T07:45:59Z
dc.identifier.doi10.1186/s12864-016-2739-6
dc.identifier.cristin1368527


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Navngivelse 3.0 Norge
Except where otherwise noted, this item's license is described as Navngivelse 3.0 Norge