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dc.contributor.authorStenberg, Ulf Simon
dc.contributor.authorLi, Jing
dc.contributor.authorGjuvsland, Arne Bjørke
dc.contributor.authorPersson, Karl
dc.contributor.authorDemitz-Helin, Erik
dc.contributor.authorGonzalez-Pena, Carles
dc.contributor.authorYue, Jia-Xing
dc.contributor.authorGilchrist, Ciaran
dc.contributor.authorÄrengård, Timmy
dc.contributor.authorGhiaci, Payam
dc.contributor.authorLarsson-Berglund, Lisa
dc.contributor.authorZackrisson, Martin
dc.contributor.authorSmits, Silvana
dc.contributor.authorHallin, Johan
dc.contributor.authorHöög, Johanna L.
dc.contributor.authorMolin, Mikael
dc.contributor.authorLiti, Gianni
dc.contributor.authorOmholt, Stig William
dc.contributor.authorWarringer, Jonas
dc.date.accessioned2023-03-30T11:50:17Z
dc.date.available2023-03-30T11:50:17Z
dc.date.created2022-10-04T11:00:14Z
dc.date.issued2022
dc.identifier.citationeLIFE. 2022, 11 .
dc.identifier.issn2050-084X
dc.identifier.urihttps://hdl.handle.net/11250/3061148
dc.language.isoeng
dc.titleGenetically controlled mtDNA deletions prevent ROS damage by arresting oxidative phosphorylation
dc.title.alternativeGenetically controlled mtDNA deletions prevent ROS damage by arresting oxidative phosphorylation
dc.typePeer reviewed
dc.typeJournal article
dc.description.versionpublishedVersion
dc.source.pagenumber0
dc.source.volume11
dc.source.journaleLIFE
dc.identifier.doi10.7554/elife.76095
dc.identifier.cristin2058254
cristin.ispublishedtrue
cristin.fulltextoriginal
cristin.qualitycode2


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